Apoptosis is a programmed cell death that occurs in multicellular organisms for various reasons, such as DNA damage, organismal needs, or developmental processes. Learn about the two major types of apoptosis pathways, the role of apoptosis in cancer prevention, and the examples of apoptosis in frogs, humans, and mice.

Apoptosis is the normal, planned death of damaged cells in your body. Learn how apoptosis works, why it’s important for your health, and how it can go wrong in certain conditions.

Apoptosis is a mechanism that allows cells to self-destruct when stimulated by various factors. Learn about the history, the molecular pathways, and the medical significance of apoptosis from Britannica.

In multicellular organisms, cells that are no longer needed or are a threat to the organism are destroyed by a tightly regulated cell suicide process known as programmed cell death, or apoptosis. Apoptosis is mediated by proteolytic enzymes called caspases, which trigger cell death by cleaving specific proteins in the cytoplasm and nucleus.

Apoptosis is a peer-reviewed journal that publishes original and review articles on the mechanisms and roles of apoptosis in various human diseases. It also covers topics such as PANoptosis, autophagy, cancer, autoimmunity, and ageing.

Apoptosis is the programmed cell death that occurs during development and in adults to eliminate damaged or unwanted cells. Learn how apoptosis works, its role in preventing cancer and neurodegenerative diseases, and its origin and meaning.

Apoptosis is a highly regulated and controlled process that unfolds in distinct stages, each characterized by specific cellular changes and biochemical events. Initiation: Internal or external signals trigger the apoptosis pathways. Early Apoptosis: Key indicators include cell shrinkage, chromatin condensation, and membrane blebbing.